Project Information
Conservation and divergence of YODA MAPKKK functionin regulation of grass epidermal patterning
All multicellular organisms must properly pattern cell types to generate functional tissues and organs. The organized and predictable cell lineages of the Brachypodium leaf enabled us to characterize the role of the MAPK kinase kinase gene BdYODA1 in regulating asymmetric cell divisions. We find that YODA genes promote normal stomatal spacing patterns in both Arabidopsis and Brachypodium, despite species-specific differences in those patterns. Using lineage tracing and cell fate markers, we show that, unexpectedly, patterning defects in bdyoda1 mutants do not arise from faulty physical asymmetry in cell divisions but rather from improper enforcement of alternative cellular fates after division. These cross-species comparisons allow us to refine our understanding of MAPK activities during plant asymmetric cell divisions.Lab: Bergmann Lab, Department of Biology, Stanford University, Stanford, CA 94305-5020, USA.
Contact: Dominique Bergmann, dbergmann@stanford.edu
List of Transformation
| Transformation | Description | Genetic Background | Construct | Publication |
|---|---|---|---|---|
| BdRT0001 | transformation BdRT0001 to Bd21-3 | Bd21-3 | BdTG0001-1; BdTG0001-1 | Abrash et al., (2018). Conservation and divergence of YODA MAPKKK function in regulation of grass epidermal patterning. Development 145, dev165860. doi:10.1242/dev.165860 |
This database is supported by NSF (IOS-1546625) and hosted by BTI.
